{"product_id":"male-cardiometabolic-hormone-profile","title":"Male CardioMetabolic \u0026 Hormone Profile | Specialist Doctor Report Included","description":"\u003cp\u003e\u003cstrong\u003eTest ID: SB-645 | Saliva 4-Point Cortisol + Blood Spot LCMS Hormones 7 with SHBG \u0026amp; CardioMetabolic Panel\u003c\/strong\u003e\u003c\/p\u003e\n\u003cp\u003eThe \u003cstrong\u003eMale CardioMetabolic \u0026amp; Hormone Profile\u003c\/strong\u003e is an advanced at-home health assessment for men measuring \u003cstrong\u003e18 key hormones and markers\u003c\/strong\u003e across three interconnected systems: the full daily cortisol stress rhythm by 4-point saliva testing, a complete blood spot LCMS sex hormone panel including SHBG, and a six-marker cardiometabolic panel. It combines the \u003cstrong\u003e4-point saliva cortisol awakening response and diurnal rhythm\u003c\/strong\u003e with \u003cstrong\u003eblood spot LCMS sex hormones\u003c\/strong\u003e, \u003cstrong\u003eSHBG\u003c\/strong\u003e, and \u003cstrong\u003efasting insulin\u003c\/strong\u003e — providing the hormonal and metabolic picture that standard NHS testing cannot achieve without thyroid assessment.\u003c\/p\u003e\n\u003cp\u003eIn men, testosterone, cortisol, SHBG, and insulin are mechanistically inseparable. The morning cortisol awakening response sets Leydig cell testosterone output, hepatic SHBG synthesis, and insulin sensitivity for the entire day. Chronic cortisol elevation depletes testosterone via pregnenolone steal and direct Leydig cell suppression, simultaneously elevates SHBG to inactivate the testosterone that remains, and drives insulin resistance, visceral fat accumulation, triglyceride elevation, and hsCRP. The result is a man with normal total testosterone, elevated SHBG, suppressed free testosterone, insulin resistance, and cardiovascular inflammation — all caused by the same cortisol pattern, but invisible without measuring all three systems at once.\u003c\/p\u003e\n\u003cp\u003eTest from home with confidence — no clinic visit required. Results within \u003cstrong\u003e3–5 working days\u003c\/strong\u003e.\u003c\/p\u003e\n\u003chr\u003e\n\u003ch2\u003eWhy Cortisol Rhythm, SHBG, and CardioMetabolic Health Must Be Assessed Together in Men\u003c\/h2\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eThe 4-point cortisol rhythm determines free testosterone availability via SHBG in men\u003c\/strong\u003e — cortisol elevation at any time point — morning, noon, evening, or night — triggers hepatic SHBG synthesis, which binds and inactivates testosterone regardless of how much the testes produce; a blunted morning cortisol awakening response impairs Leydig cell stimulation; elevated noon cortisol drives aromatase activity, raising oestradiol and further elevating SHBG; elevated night cortisol suppresses overnight testosterone production; only four-time-point measurement reveals which phase of the day is driving the SHBG-mediated testosterone deficiency and cardiometabolic deterioration\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eSHBG is the most commonly missed cause of low testosterone symptoms in men with “normal” blood results\u003c\/strong\u003e — total testosterone measured without SHBG cannot distinguish between a man with adequate free testosterone and a man with all testosterone bound and inactivated by elevated SHBG; elevated SHBG — driven by cortisol, oestradiol excess, age, caloric restriction, and hepatic dysfunction — produces full androgenic deficiency symptoms including fatigue, low libido, erectile dysfunction, poor recovery, and brain fog in men whose total testosterone falls within the standard reference range\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eCortisol drives every cardiometabolic risk marker simultaneously in men\u003c\/strong\u003e — through hepatic insulin receptor dysregulation, cortisol causes insulin resistance, raising fasting insulin years before HbA1c becomes abnormal; through visceral fat promotion, it drives aromatase-mediated oestradiol conversion, SHBG elevation, and HPG axis suppression; through hepatic lipid dysregulation, it raises triglycerides and suppresses HDL; and through all these pathways simultaneously, it elevates hsCRP — the convergence marker of androgenic and cardiometabolic deterioration in men\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eInsulin resistance and free testosterone decline are mutually reinforcing in men\u003c\/strong\u003e — elevated insulin promotes visceral fat and aromatase-driven testosterone-to-oestradiol conversion, raising SHBG and suppressing the HPG axis; low free testosterone worsens insulin sensitivity, promoting further visceral fat; fasting insulin is the earliest detectable marker of this cycle — elevated years before HbA1c or fasting glucose becomes abnormal — and directly linked to erectile dysfunction, declining androgenic function, and rising cardiovascular risk in men\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003ehsCRP is the convergence biomarker of cortisol, SHBG-mediated testosterone deficiency, and insulin resistance in men\u003c\/strong\u003e — elevated simultaneously by cortisol at any of the four time points, low free testosterone, insulin resistance, visceral fat, and oestradiol excess; a stronger independent predictor of cardiovascular events and all-cause mortality in men than LDL cholesterol; and directly elevated by the same cortisol-SHBG-insulin mechanism this panel is designed to characterise\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eWhat This Test Measures — 18 Markers Across Three Systems\u003c\/h2\u003e\n\u003ch3\u003eSaliva — 4-Point Cortisol Profile (4 markers)\u003c\/h3\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eMorning Cortisol (Cortisol Awakening Response):\u003c\/strong\u003e Sets Leydig cell testosterone output, hepatic SHBG synthesis rate, and insulin sensitivity for the entire day. An exaggerated morning response drives testosterone suppression, SHBG elevation, and morning insulin resistance; a blunted awakening response signals HPA burnout, reduced Leydig cell responsiveness, and impaired morning metabolic priming.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eNoon Cortisol:\u003c\/strong\u003e Elevated noon cortisol drives visceral fat deposition, aromatase-mediated testosterone-to-oestradiol conversion, SHBG elevation, triglyceride rise via hepatic lipid dysregulation, and HDL suppression — the direct mechanism by which chronic midday occupational stress accelerates androgenic decline and cardiometabolic deterioration in men during the working day.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eEvening Cortisol:\u003c\/strong\u003e Should decline significantly to allow testosterone and growth hormone to rise during early sleep cycles. Elevated evening cortisol suppresses overnight testosterone production, elevates SHBG through hepatic cortisol-SHBG coupling, maintains insulin resistance into the night, and independently raises hsCRP — producing next-day fatigue, reduced recovery, and worsening androgenic and metabolic function regardless of morning values.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eNight Cortisol:\u003c\/strong\u003e Suppresses the deep sleep phases during which the majority of daily testosterone is produced and growth hormone mediates fat metabolism. Drives overnight visceral fat accumulation, fasting insulin elevation, hsCRP rise, and aromatase-mediated oestradiol and SHBG excess — compounding every androgenic and cardiometabolic risk factor measured by the following morning’s blood spot.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003ch3\u003eBlood Spot LCMS — Sex Hormones \u0026amp; SHBG (8 markers)\u003c\/h3\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eOestradiol (E2):\u003c\/strong\u003e Present in men via aromatase conversion of testosterone. Physiologically important for bone density, cardiovascular protection, and cognitive function — but excess oestradiol from visceral fat, cortisol elevation, or insulin resistance suppresses testosterone via the HPG axis, elevates SHBG, causes gynaecomastia, raises triglycerides, and elevates cardiovascular risk. The oestradiol-to-free-testosterone ratio, calculated using SHBG, is the primary androgenic and cardiometabolic health indicator in men. Measured by blood spot LCMS for the highest available accuracy.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eOestriol (E3):\u003c\/strong\u003e Reflects oestrogen conversion pathways and aromatase activity in men. Elevated E3 indicates increased oestrogen precursor load or conversion; the E3-to-E2 ratio provides aromatase activity context beyond what E2 alone reveals.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eOestrone (E1):\u003c\/strong\u003e Produced by androgen conversion in adipose tissue. Elevated oestrone in men with visceral fat drives systemic oestrogenic load, compounds HPG suppression, elevates SHBG, and is associated with increased cardiometabolic and inflammatory risk.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eProgesterone (Pg):\u003c\/strong\u003e Present in men in small but clinically important amounts. Acts as a natural 5-alpha reductase inhibitor, blocking DHT conversion; has direct anti-oestrogenic, anti-inflammatory, and cardioprotective effects; and supports insulin sensitivity and sleep quality. Low progesterone is associated with elevated DHT, prostate risk, anxiety, and increased cardiometabolic inflammation in men.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTestosterone (T):\u003c\/strong\u003e The primary male androgen — driving energy, libido, muscle mass, bone density, insulin sensitivity, HDL maintenance, and cardiovascular health. Measured by blood spot LCMS for highest accuracy. Interpreted alongside SHBG to calculate free testosterone bioavailability — the clinically definitive androgenic assessment that testosterone measurement without SHBG cannot provide.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eDHEAS (DS):\u003c\/strong\u003e The primary adrenal androgen precursor in men, declining from the mid-30s and depleted under chronic cortisol elevation. Low DHEAS reduces testosterone precursor availability, impairs stress resilience, and has direct anti-inflammatory and insulin-sensitising effects. The 4-point cortisol rhythm reveals the HPA dysregulation mechanism driving DHEAS depletion.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eCortisol (C) — Blood Spot LCMS:\u003c\/strong\u003e The single blood spot cortisol measurement by LCMS provides a direct comparator to the 4-point saliva cortisol rhythm, enabling cross-matrix validation and the most complete cortisol assessment available in an at-home test without thyroid assessment.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eSex Hormone Binding Globulin (SHBG):\u003c\/strong\u003e The bioavailability gatekeeper for testosterone in men. Elevated SHBG — driven by cortisol elevation, oestradiol excess, age, and caloric restriction — binds and inactivates testosterone regardless of its total production, causing full androgenic deficiency symptoms in men with normal total testosterone values. SHBG is essential for calculating free testosterone, interpreting the oestradiol-testosterone ratio, and identifying whether symptoms are driven by SHBG elevation, production failure, or HPG suppression — distinctions only possible when SHBG is measured alongside the 4-point cortisol rhythm.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003ch3\u003eBlood Spot — CardioMetabolic Panel (6 markers)\u003c\/h3\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eFasting Insulin:\u003c\/strong\u003e The earliest detectable marker of insulin resistance in men — elevated years before HbA1c or fasting glucose becomes abnormal. Directly promoted by cortisol at any time point, low free testosterone (worsened by elevated SHBG), and visceral fat. Elevated fasting insulin drives aromatase activity, increases oestradiol conversion and SHBG elevation, independently raises cardiovascular risk, and is closely linked to erectile dysfunction and androgenic decline. The most actionable early metabolic marker for men with hormonal and energy symptoms.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eHbA1c:\u003c\/strong\u003e Reflects average blood sugar over 8–12 weeks. Men with chronically elevated cortisol, low free testosterone, and elevated SHBG have markedly elevated risk of pre-diabetes and type 2 diabetes; HbA1c provides the medium-term blood sugar context that anchors the fasting insulin and cortisol rhythm assessment.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eHDL Cholesterol:\u003c\/strong\u003e The protective cardiovascular fraction directly maintained by free testosterone and suppressed by SHBG-mediated testosterone deficiency, insulin resistance, and cortisol elevation — three pathways all assessed comprehensively by this panel.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTriglycerides:\u003c\/strong\u003e Elevated by cortisol at any of the four time points, insulin resistance, and oestradiol excess simultaneously. Rise before LDL changes and are a primary cardiometabolic risk marker particularly relevant for men with visceral fat, hormonal decline, and elevated SHBG.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTotal Cholesterol:\u003c\/strong\u003e Provides overall lipid context. Rising cholesterol in men with fatigue, low libido, and elevated SHBG is frequently a combined hormonal and cortisol event rather than a purely dietary one — best understood alongside the hormone levels this panel measures directly.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003ehsCRP (High-Sensitivity C-Reactive Protein):\u003c\/strong\u003e One of the strongest independent predictors of cardiovascular events and all-cause mortality in men. Elevated simultaneously by low free testosterone, SHBG-mediated androgenic deficiency, cortisol dysregulation at any time point, insulin resistance, visceral fat, and oestradiol excess — all pathways directly assessed by this panel. The convergence biomarker of the male hormonal-metabolic system.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eWho Should Consider This Test?\u003c\/h2\u003e\n\u003cp\u003eThis test is particularly relevant for men who:\u003c\/p\u003e\n\u003cul\u003e\n\u003cli\u003eHave persistent fatigue or exhaustion that does not improve with rest or sleep\u003c\/li\u003e\n\u003cli\u003eExperience unexplained weight gain or difficulty losing body fat, particularly around the abdomen\u003c\/li\u003e\n\u003cli\u003eHave low libido or reduced sexual interest\u003c\/li\u003e\n\u003cli\u003eExperience erectile dysfunction or reduced sexual performance\u003c\/li\u003e\n\u003cli\u003eHave poor recovery from exercise, training, or sustained high workload\u003c\/li\u003e\n\u003cli\u003eExperience brain fog, poor concentration, or reduced cognitive sharpness\u003c\/li\u003e\n\u003cli\u003eHave low mood, reduced motivation, or mild depression\u003c\/li\u003e\n\u003cli\u003eExperience sugar cravings, energy crashes, or afternoon slumps\u003c\/li\u003e\n\u003cli\u003eHave elevated cholesterol, triglycerides, or blood sugar on previous testing\u003c\/li\u003e\n\u003cli\u003eStruggle with insomnia, night waking, or unrefreshing sleep\u003c\/li\u003e\n\u003cli\u003eAre under chronic stress, burnout, or sustained high-pressure workload\u003c\/li\u003e\n\u003cli\u003eHave been told their testosterone is “normal” but continue to experience androgenic symptoms (elevated SHBG is frequently the explanation)\u003c\/li\u003e\n\u003cli\u003eAre currently on TRT and want to monitor SHBG, oestradiol, and cardiometabolic markers alongside testosterone\u003c\/li\u003e\n\u003cli\u003eHave a family history of heart disease, diabetes, or metabolic syndrome\u003c\/li\u003e\n\u003cli\u003eAre men over 35 seeking a comprehensive androgenic, stress-rhythm, and cardiometabolic health baseline without thyroid assessment\u003c\/li\u003e\n\u003cli\u003eWant the full cortisol-SHBG-testosterone-insulin picture before deciding whether to add thyroid testing\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eKey Features\u003c\/h2\u003e\n\u003cul\u003e\n\u003cli\u003e18-marker tri-system panel: 4-point saliva cortisol rhythm, blood spot LCMS sex hormones + SHBG, and full cardiometabolic panel\u003c\/li\u003e\n\u003cli\u003e4-point cortisol profile — morning, noon, evening, and night — the only method that reveals the full daily cortisol rhythm and its SHBG-mediated impact on free testosterone and cardiometabolic health\u003c\/li\u003e\n\u003cli\u003eBlood spot LCMS sex hormones — gold standard accuracy across the full androgenic range\u003c\/li\u003e\n\u003cli\u003eSHBG included — essential for calculating free testosterone and identifying the mechanism of androgenic deficiency in men with normal total testosterone\u003c\/li\u003e\n\u003cli\u003eAll three oestrogens (E1, E2, E3) for complete oestrogenic load and aromatase activity assessment\u003c\/li\u003e\n\u003cli\u003eSix cardiometabolic markers — fasting insulin, HbA1c, HDL, triglycerides, total cholesterol, hsCRP\u003c\/li\u003e\n\u003cli\u003eResults within \u003cstrong\u003e3–5 working days\u003c\/strong\u003e after lab receipt\u003c\/li\u003e\n\u003cli\u003eExpert analysis by a \u003cstrong\u003eHormone Specialist PhD Doctor\u003c\/strong\u003e\n\u003c\/li\u003e\n\u003cli\u003eClear graphical and numerical results with personalised commentary\u003c\/li\u003e\n\u003cli\u003eAll-inclusive price — laboratory fees included, no hidden costs\u003c\/li\u003e\n\u003cli\u003eValid for 12 months from purchase\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eHow It Works\u003c\/h2\u003e\n\u003col\u003e\n\u003cli\u003e\n\u003cstrong\u003eOrder the kit online\u003c\/strong\u003e — delivered directly to your door.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eCollect your saliva samples\u003c\/strong\u003e at four time points (morning, noon, evening, night) for the cortisol rhythm panel, and your \u003cstrong\u003eblood spot sample\u003c\/strong\u003e via a simple finger-prick for the LCMS hormone, SHBG, and cardiometabolic panels, following the detailed instructions. Morning saliva should be collected before 9am for optimal cortisol awakening response accuracy.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003ePost your samples to the lab\u003c\/strong\u003e using the return instructions in your kit.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eReceive your results within 3–5 working days\u003c\/strong\u003e with specialist commentary and clear next-step guidance.\u003c\/li\u003e\n\u003c\/ol\u003e\n\u003chr\u003e\n\u003ch2\u003eWhat Your Report Includes\u003c\/h2\u003e\n\u003cul\u003e\n\u003cli\u003eAll 18 marker results with male-specific reference ranges\u003c\/li\u003e\n\u003cli\u003eVisual cortisol rhythm chart showing your full four-point daily curve\u003c\/li\u003e\n\u003cli\u003eVisual charts for all six sex hormones, SHBG, and all six cardiometabolic markers\u003c\/li\u003e\n\u003cli\u003eIntegrated clinical interpretation of how your cortisol rhythm, testosterone, SHBG, oestradiol, and cardiometabolic results interact as a single male hormonal-metabolic system\u003c\/li\u003e\n\u003cli\u003eSHBG-adjusted free testosterone bioavailability assessment — the definitive androgenic status calculation\u003c\/li\u003e\n\u003cli\u003eOestradiol-to-free-testosterone ratio analysis in SHBG and cortisol context\u003c\/li\u003e\n\u003cli\u003eCortisol-SHBG coupling assessment — directly mapping each cortisol time point to SHBG elevation and free testosterone suppression\u003c\/li\u003e\n\u003cli\u003eInsulin resistance pattern assessment (fasting insulin + HbA1c)\u003c\/li\u003e\n\u003cli\u003eCardiovascular risk profile in full androgenic and SHBG hormonal context (HDL, triglycerides, cholesterol, hsCRP)\u003c\/li\u003e\n\u003cli\u003eSpecialist commentary by a Hormone Specialist PhD Doctor\u003c\/li\u003e\n\u003cli\u003ePersonalised next-step recommendations\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eFrequently Asked Questions\u003c\/h2\u003e\n\u003ch3\u003eWhat does this test measure?\u003c\/h3\u003e\n\u003cp\u003e4-point saliva cortisol (morning, noon, evening, night) + Oestradiol (E2), Oestriol (E3), Oestrone (E1), Progesterone, Testosterone, DHEAS, Cortisol, SHBG (blood spot LCMS) + Fasting Insulin, HbA1c, HDL Cholesterol, Triglycerides, Total Cholesterol, hsCRP (blood spot) — 18 markers in total.\u003c\/p\u003e\n\u003ch3\u003eDoes this test include thyroid assessment?\u003c\/h3\u003e\n\u003cp\u003eNo. This profile assesses cortisol rhythm, sex hormones, SHBG, and cardiometabolic markers — providing the complete androgenic and metabolic picture without thyroid markers. For men who want thyroid assessment alongside this panel, the \u003cstrong\u003eMale Advanced Hormone, Thyroid \u0026amp; CardioMetabolic Test (SB-649M)\u003c\/strong\u003e adds a full 6-marker thyroid panel including TPO and Tg antibodies with a single cortisol measurement, and the \u003cstrong\u003eMale Comprehensive Hormone, Thyroid \u0026amp; CardioMetabolic Profile (SB-646M)\u003c\/strong\u003e adds the full thyroid panel alongside the 4-point cortisol rhythm and SHBG.\u003c\/p\u003e\n\u003ch3\u003eWhy do I need SHBG tested alongside my testosterone?\u003c\/h3\u003e\n\u003cp\u003eSHBG binds and inactivates testosterone in the bloodstream. A man with normal total testosterone but elevated SHBG — caused by cortisol, oestradiol excess, age, or caloric restriction — has full symptoms of androgen deficiency despite a “normal” result. SHBG is the most common reason men are told their testosterone is normal but continue to experience fatigue, low libido, and poor recovery. This panel identifies both the SHBG level and, through the 4-point cortisol rhythm, the most likely mechanism driving it.\u003c\/p\u003e\n\u003ch3\u003eWhy is a 4-point cortisol rhythm more informative than a single cortisol measurement?\u003c\/h3\u003e\n\u003cp\u003eA single cortisol value cannot identify which part of the day is driving androgenic or cardiometabolic deterioration. A blunted morning awakening response impairs Leydig cell testosterone output all day; elevated noon cortisol drives aromatase and SHBG; elevated evening cortisol suppresses overnight testosterone; night cortisol elevation directly reduces the sleep phases during which testosterone is produced. Each pattern has different hormonal and metabolic consequences — and different intervention targets.\u003c\/p\u003e\n\u003ch3\u003eCan I use this test if I am on TRT?\u003c\/h3\u003e\n\u003cp\u003eYes. This test is particularly valuable for men on TRT who want to monitor SHBG, oestradiol, cortisol rhythm, and cardiometabolic markers alongside testosterone. TRT increases aromatisation and raises oestradiol, which elevates SHBG and can reduce the free testosterone benefit of treatment. The 4-point cortisol rhythm identifies whether cortisol management would improve TRT efficacy and reduce cardiometabolic inflammation.\u003c\/p\u003e\n\u003ch3\u003eWhen will I get my results?\u003c\/h3\u003e\n\u003cp\u003eWithin \u003cstrong\u003e3–5 working days\u003c\/strong\u003e after your samples reach the laboratory.\u003c\/p\u003e\n\u003ch3\u003eAre there hidden costs?\u003c\/h3\u003e\n\u003cp\u003eNo. Laboratory analysis and specialist review are fully included. You are responsible for postage to the lab.\u003c\/p\u003e\n\u003ch3\u003eHow long is the kit valid?\u003c\/h3\u003e\n\u003cp\u003e12 months from purchase.\u003c\/p\u003e\n\u003chr\u003e\n\u003ch2\u003eWhy We Partner with ZRT Laboratory\u003c\/h2\u003e\n\u003cp\u003eAt Hormone Lab UK, every home test kit is analysed by \u003cstrong\u003eZRT Laboratory\u003c\/strong\u003e, a globally recognised, \u003cstrong\u003eCLIA-certified\u003c\/strong\u003e diagnostic and research laboratory specialising in hormone testing, thyroid testing, fertility testing, adrenal function, neurotransmitter analysis, cardiometabolic health, heavy metals, and wellness testing. Founded in 1998 by renowned biochemist and breast cancer researcher \u003cstrong\u003eDr. David Zava, PhD\u003c\/strong\u003e, ZRT has become one of the world’s leading laboratories for advanced functional health testing.\u003c\/p\u003e\n\u003cp\u003eWith \u003cstrong\u003emore than 11 million laboratory tests\u003c\/strong\u003e performed and healthcare professionals in \u003cstrong\u003eover 96 countries\u003c\/strong\u003e relying on its expertise, ZRT is recognised for scientific excellence, analytical accuracy, and continuous innovation. The laboratory pioneered \u003cstrong\u003eat-home saliva hormone testing\u003c\/strong\u003e, introduced the \u003cstrong\u003efirst commercially available dried blood spot (DBS) hormone tests\u003c\/strong\u003e, and developed \u003cstrong\u003eDried Urine Testing (DUTS)\u003c\/strong\u003e, helping make advanced laboratory diagnostics more accessible and clinically meaningful.\u003c\/p\u003e\n\u003cp\u003eEvery sample is analysed using state-of-the-art \u003cstrong\u003eLC-MS\/MS\u003c\/strong\u003e and \u003cstrong\u003eICP-MS\u003c\/strong\u003e technology under rigorous quality control standards. ZRT collaborates with the \u003cstrong\u003eCDC\u003c\/strong\u003e, \u003cstrong\u003eNIH\u003c\/strong\u003e, and leading universities, supporting ongoing research and advances in laboratory medicine.\u003c\/p\u003e","brand":"Hormone Lab UK","offers":[{"title":"Default Title","offer_id":57961607725437,"sku":"1020","price":419.0,"currency_code":"GBP","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/1885\/4859\/files\/SalivaandBloodSpotHormoneTestKit.webp?v=1785073903","url":"https:\/\/hormonelab.co.uk\/products\/male-cardiometabolic-hormone-profile","provider":"Hormone Lab UK","version":"1.0","type":"link"}