{"product_id":"male-comprehensive-hormone-thyroid-cardiometabolic-test","title":"Male Comprehensive Hormone, Thyroid \u0026 CardioMetabolic Test | Specialist Doctor Report Included","description":"\u003cp\u003e\u003cstrong\u003eTest ID: SB-652 | Saliva LCMS Hormones 7 with 4-Point Cortisol + Blood Spot Full Thyroid \u0026amp; CardioMetabolic Panel\u003c\/strong\u003e\u003c\/p\u003e\n\u003cp\u003eThe \u003cstrong\u003eMale Comprehensive Hormone, Thyroid \u0026amp; CardioMetabolic Test\u003c\/strong\u003e is the most complete at-home health profile available for men anywhere in the UK, measuring \u003cstrong\u003e22 key hormones and markers\u003c\/strong\u003e across four interconnected systems in a single kit: androgenic and sex hormone balance, the full daily cortisol stress rhythm, complete thyroid function including autoimmune antibodies, and a comprehensive cardiometabolic panel. It uses \u003cstrong\u003esaliva LCMS (Liquid Chromatography–Mass Spectrometry)\u003c\/strong\u003e for sex hormones and cortisol — the gold standard for bioavailable hormone measurement — combined with a \u003cstrong\u003efull blood spot thyroid and cardiometabolic panel\u003c\/strong\u003e.\u003c\/p\u003e\n\u003cp\u003eThis test exists because male health does not divide neatly into separate systems. In men, testosterone, cortisol, thyroid hormones, and insulin form a single integrated regulatory network — one in which disruption to any system cascades through all the others. Cortisol depletes testosterone through pregnenolone steal and Leydig cell suppression, drives visceral fat and aromatase-driven oestradiol conversion, impairs thyroid T3 conversion, and causes insulin resistance — all simultaneously. Hypothyroidism directly suppresses testosterone synthesis, compounds every symptom of androgen decline, and is missed by TSH alone in the presence of cortisol-driven T3 suppression. Insulin resistance promotes aromatase activity and further testosterone decline. And elevated oestradiol feeds back to suppress the hypothalamic-pituitary-gonadal axis, completing the cycle. No standard blood panel tests all four systems together. This one does.\u003c\/p\u003e\n\u003cp\u003eTest from home with confidence — no clinic visit required. Results within \u003cstrong\u003e3–5 working days\u003c\/strong\u003e.\u003c\/p\u003e\n\u003chr\u003e\n\u003ch2\u003eThe Male Hormonal-Metabolic System: Why All Four Must Be Tested Together\u003c\/h2\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eTestosterone is the central coordinator of male metabolic and cardiovascular health\u003c\/strong\u003e — testosterone maintains insulin sensitivity, HDL cholesterol, lean muscle mass, bone density, and vascular health; low testosterone independently raises triglycerides, suppresses HDL, promotes visceral fat, elevates hsCRP, and increases the risk of metabolic syndrome, type 2 diabetes, and cardiovascular disease; at the same time, metabolic dysfunction further suppresses testosterone through aromatase and HPA axis dysregulation — a compounding cycle that single-system tests cannot interrupt\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eCortisol is the primary suppressor of testosterone and the primary driver of male cardiometabolic deterioration\u003c\/strong\u003e — through pregnenolone steal, direct Leydig cell suppression, and HPA axis dysregulation, chronic cortisol depletes testosterone while simultaneously driving visceral fat, aromatase-driven oestradiol excess, insulin resistance, triglyceride elevation, and hsCRP — a five-way attack that only the combined 4-panel test can fully characterise and explain\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eThyroid dysfunction is a major and systematically missed cause of low testosterone in men\u003c\/strong\u003e — hypothyroidism directly suppresses Leydig cell testosterone production and impairs the protein metabolism, recovery, and energy generation that testosterone depends on; cortisol-driven T3 suppression produces the same hypothyroid symptom picture with a normal TSH; Hashimoto’s thyroiditis is present in men for years before TSH becomes abnormal; and TSH alone misses all three of these patterns — making the full thyroid panel including FT3, FT4, TT4, TPO, and Tg antibodies essential for any man with unresolved fatigue, weight gain, or low testosterone symptoms\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eInsulin resistance and testosterone decline are mutually reinforcing in men\u003c\/strong\u003e — elevated insulin promotes visceral fat and aromatase-driven oestrogen conversion; low testosterone worsens insulin sensitivity; together they drive metabolic syndrome, type 2 diabetes, and erectile dysfunction simultaneously; fasting insulin is the earliest detectable marker of this process — elevated years before HbA1c or fasting glucose becomes abnormal — and cannot be assessed by any standard NHS hormone panel\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003ehsCRP is the convergence marker of the entire male hormonal-metabolic axis\u003c\/strong\u003e — elevated by low testosterone, cortisol dysregulation, insulin resistance, visceral fat, oestradiol excess, and Hashimoto’s autoimmunity simultaneously; it is a stronger independent predictor of cardiovascular events and all-cause mortality in men than LDL cholesterol, and is directly modifiable through integrated hormonal, thyroid, and metabolic intervention\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eWhat This Test Measures — 22 Markers Across Four Systems\u003c\/h2\u003e\n\u003ch3\u003eSaliva LCMS — Sex Hormones (6 markers)\u003c\/h3\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eOestradiol (E2):\u003c\/strong\u003e Present in men via aromatase conversion of testosterone. Essential for bone density, cardiovascular protection, and cognitive function — but excess oestradiol from visceral fat, cortisol, or alcohol suppresses testosterone via the HPG axis, causes gynaecomastia, raises triglycerides, and elevates cardiovascular risk. The oestradiol-to-testosterone ratio is a primary androgenic and cardiometabolic health indicator in men. Measured by LCMS for the free, bioavailable fraction.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eOestriol (E3):\u003c\/strong\u003e Reflects oestrogen conversion pathways and aromatase activity in men. Elevated E3 indicates increased oestrogen precursor load or conversion; the E3-to-E2 ratio provides metabolic oestrogen context beyond what E2 alone reveals.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eOestrone (E1):\u003c\/strong\u003e Produced by androgen conversion in adipose tissue. Elevated oestrone in men with visceral fat or metabolic dysfunction drives systemic oestrogenic load, compounds testosterone suppression, and is associated with increased cardiometabolic and inflammatory risk. A key indicator of adipose-driven hormonal disruption in men.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eProgesterone (Pg):\u003c\/strong\u003e Present in men in small but clinically important amounts. Acts as a natural 5-alpha reductase inhibitor, blocking DHT conversion; has direct anti-oestrogenic, anti-inflammatory, neuroprotective, and cardioprotective effects; supports insulin sensitivity, sleep quality, and thyroid function. Low progesterone in men is associated with elevated DHT, prostate risk, anxiety, cardiometabolic inflammation, and poor sleep architecture.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTestosterone (T):\u003c\/strong\u003e The primary male androgen — responsible for energy, libido, muscle mass, bone density, insulin sensitivity, HDL maintenance, and cardiovascular health. Saliva LCMS measures the free, bioavailable fraction — the clinically relevant value that drives androgenic function and correlates with symptoms, as opposed to total blood testosterone which includes the SHBG-bound, inactive fraction that can mask significant free testosterone deficiency.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eDHEAS (DS):\u003c\/strong\u003e The primary adrenal androgen precursor in men, declining from the mid-30s and depleted under chronic stress or overtraining. Low DHEAS reduces testosterone precursor availability, impairs stress resilience, and has direct anti-inflammatory and insulin-sensitising effects that protect against cardiometabolic deterioration. Elevated DHEAS under adrenal overactivation drives androgen excess and inflammatory cardiovascular risk.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003ch3\u003eSaliva — 4-Point Cortisol Profile (4 markers)\u003c\/h3\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eMorning Cortisol:\u003c\/strong\u003e The Cortisol Awakening Response — sets testosterone availability, LH pulsatility, insulin sensitivity, and thyroid activation for the day. An exaggerated response drives morning testosterone suppression and insulin resistance; a blunted response signals HPA burnout, impaired thyroid activation, and metabolic underperformance from the first hours.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eNoon Cortisol:\u003c\/strong\u003e Elevated noon cortisol promotes visceral fat, insulin resistance, and aromatase-driven testosterone-to-oestradiol conversion. Simultaneously impairs thyroid T4-to-T3 conversion, raises triglycerides via hepatic lipid dysregulation, and suppresses HDL — the direct mechanism by which chronic midday stress drives cardiometabolic and androgenic deterioration in men.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eEvening Cortisol:\u003c\/strong\u003e Should decline significantly to allow testosterone and growth hormone to rise during early sleep cycles. Elevated evening cortisol suppresses overnight testosterone production, elevates SHBG through hepatic cortisol-SHBG coupling, impairs thyroid cellular uptake, and maintains the insulin-resistant, pro-inflammatory state into the night.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eNight Cortisol:\u003c\/strong\u003e Suppresses the deep sleep phases during which the majority of daily testosterone is produced and growth hormone mediates fat metabolism. Drives overnight visceral fat accumulation, fasting insulin elevation, hsCRP rise, and aromatase excess — and worsens Hashimoto’s autoimmune activity through sleep-deprivation-mediated immune dysregulation.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003ch3\u003eBlood Spot — Full Thyroid Panel (6 markers)\u003c\/h3\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eTSH:\u003c\/strong\u003e Essential first-line thyroid marker — but in men, TSH alone misses cortisol-driven T3 suppression, early thyroid insufficiency, and Hashimoto’s autoimmunity present years before TSH becomes abnormal. Necessary but not sufficient.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eFree T3 (FT3):\u003c\/strong\u003e The active thyroid hormone that drives metabolism, energy, testosterone synthesis support, recovery, and cognitive function in men. Low FT3 with normal TSH is the hallmark of cortisol-driven thyroid suppression — the most commonly missed hormonal pattern in men under chronic occupational or psychological stress.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eFree T4 (FT4):\u003c\/strong\u003e The thyroid’s primary output hormone. Low FT4 with normal TSH indicates early thyroid insufficiency or impaired peripheral conversion — often preceding symptomatic hypothyroidism and testosterone suppression by years.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTotal T4 (TT4):\u003c\/strong\u003e Provides full T4 context including the bound fraction — important interpretive context for men on TRT where thyroid-binding proteins may be affected.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTPO Antibodies (Anti-TPO):\u003c\/strong\u003e The primary diagnostic marker for Hashimoto’s thyroiditis in men. Frequently elevated years before TSH becomes abnormal; associated with progressive thyroid decline, Leydig cell testosterone suppression, fatigue, and metabolic deterioration. Directly worsened by insulin resistance and cortisol dysregulation — both assessed by this test.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eThyroglobulin Antibodies (Anti-Tg):\u003c\/strong\u003e Essential second Hashimoto’s marker. Some men have elevated Tg antibodies with normal TPO — making both necessary for complete autoimmune thyroid assessment and early detection of progressive thyroid-driven testosterone suppression.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003ch3\u003eBlood Spot — CardioMetabolic Panel (6 markers)\u003c\/h3\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eFasting Insulin:\u003c\/strong\u003e The earliest detectable marker of insulin resistance in men — elevated years before HbA1c becomes abnormal. Directly promoted by cortisol excess, low testosterone, and visceral fat. Elevated fasting insulin drives aromatase activity, increases oestrogen conversion, independently raises cardiovascular risk, and is closely linked to erectile dysfunction and testosterone decline. The most actionable early metabolic marker for men with androgenic symptoms.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eHbA1c:\u003c\/strong\u003e Reflects average blood sugar over 8–12 weeks. Men with chronically elevated cortisol, low testosterone, and hypothyroidism have markedly elevated risk of pre-diabetes and type 2 diabetes; HbA1c provides the medium-term blood sugar context that anchors the fasting insulin result.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eHDL Cholesterol:\u003c\/strong\u003e The protective cardiovascular fraction directly maintained by testosterone. HDL falls with low testosterone, insulin resistance, cortisol elevation, and hypothyroidism — four pathways all assessed by this test. A primary cardiometabolic marker of androgenic decline in men.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTriglycerides:\u003c\/strong\u003e Elevated by cortisol, insulin resistance, oestradiol excess, and hypothyroidism simultaneously — all four assessed here. Rise before LDL changes and are a primary cardiometabolic risk marker for men with visceral fat, hormonal decline, and thyroid dysfunction.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eTotal Cholesterol:\u003c\/strong\u003e Directly elevated by hypothyroidism and low testosterone. Rising cholesterol in men with fatigue and low libido is frequently a hormonal and thyroid event rather than a purely dietary one — requiring the integrated assessment this panel provides.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003ehsCRP (High-Sensitivity C-Reactive Protein):\u003c\/strong\u003e One of the strongest independent predictors of cardiovascular events and all-cause mortality in men. Elevated simultaneously by low testosterone, cortisol dysregulation, insulin resistance, visceral fat, oestradiol excess, and Hashimoto’s autoimmunity — all six pathways directly assessed by this panel. The convergence biomarker of the male hormonal-metabolic system.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eWho Should Consider This Test?\u003c\/h2\u003e\n\u003cp\u003eThis test is the ideal choice for men who experience any combination of:\u003c\/p\u003e\n\u003cul\u003e\n\u003cli\u003ePersistent fatigue or exhaustion that does not improve with sleep or rest\u003c\/li\u003e\n\u003cli\u003eUnexplained weight gain or difficulty losing body fat, particularly around the abdomen\u003c\/li\u003e\n\u003cli\u003eLow libido or reduced sexual interest\u003c\/li\u003e\n\u003cli\u003eErectile dysfunction or reduced sexual performance\u003c\/li\u003e\n\u003cli\u003ePoor recovery from exercise, training, or sustained high workload\u003c\/li\u003e\n\u003cli\u003eBrain fog, poor concentration, or reduced cognitive sharpness\u003c\/li\u003e\n\u003cli\u003eLow mood, reduced motivation, or mild depression\u003c\/li\u003e\n\u003cli\u003eFeeling cold, temperature sensitivity, or slow metabolism\u003c\/li\u003e\n\u003cli\u003eDry skin, hair thinning, or slow wound healing\u003c\/li\u003e\n\u003cli\u003eAcne, scalp hair loss, or gynaecomastia\u003c\/li\u003e\n\u003cli\u003eSugar cravings, energy crashes, or afternoon slumps\u003c\/li\u003e\n\u003cli\u003eElevated cholesterol, triglycerides, or blood sugar on previous testing\u003c\/li\u003e\n\u003cli\u003eInsomnia, night waking, or unrefreshing sleep\u003c\/li\u003e\n\u003cli\u003eChronic stress, burnout, or sustained high-pressure workload\u003c\/li\u003e\n\u003cli\u003eSymptoms of low testosterone with normal or inconclusive standard blood results\u003c\/li\u003e\n\u003cli\u003eCurrently on TRT and wanting a comprehensive hormonal, thyroid, and metabolic picture\u003c\/li\u003e\n\u003cli\u003eDiagnosed or suspected Hashimoto’s thyroiditis or autoimmune thyroid disease\u003c\/li\u003e\n\u003cli\u003eFamily history of thyroid disease, heart disease, diabetes, or metabolic syndrome\u003c\/li\u003e\n\u003cli\u003eMen over 35 seeking the single most comprehensive at-home androgenic and metabolic health assessment available\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eKey Features\u003c\/h2\u003e\n\u003cul\u003e\n\u003cli\u003e22-marker quad-system panel: saliva LCMS sex hormones, 4-point cortisol, full blood spot thyroid panel, and full cardiometabolic panel — the most comprehensive at-home male health test available in the UK\u003c\/li\u003e\n\u003cli\u003eSaliva LCMS — gold standard for free, bioavailable testosterone and sex hormone measurement in men\u003c\/li\u003e\n\u003cli\u003eAll three oestrogens (E1, E2, E3) for complete oestrogenic load and aromatase activity assessment\u003c\/li\u003e\n\u003cli\u003eFull 6-marker thyroid panel including TPO and Tg autoimmune antibodies\u003c\/li\u003e\n\u003cli\u003e4-point cortisol profile — the essential lens for interpreting all other results\u003c\/li\u003e\n\u003cli\u003eFull 6-marker cardiometabolic panel including fasting insulin and hsCRP\u003c\/li\u003e\n\u003cli\u003eResults within \u003cstrong\u003e3–5 working days\u003c\/strong\u003e after lab receipt\u003c\/li\u003e\n\u003cli\u003eExpert analysis by a \u003cstrong\u003eHormone Specialist PhD Doctor\u003c\/strong\u003e\n\u003c\/li\u003e\n\u003cli\u003eClear graphical and numerical results with personalised commentary\u003c\/li\u003e\n\u003cli\u003eAll-inclusive price — laboratory fees included, no hidden costs\u003c\/li\u003e\n\u003cli\u003eValid for 12 months from purchase\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eHow It Works\u003c\/h2\u003e\n\u003col\u003e\n\u003cli\u003e\n\u003cstrong\u003eOrder the kit online\u003c\/strong\u003e — delivered directly to your door.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eCollect your saliva samples\u003c\/strong\u003e at four time points (morning, noon, evening, night) for the sex hormone and cortisol panel, and your \u003cstrong\u003eblood spot sample\u003c\/strong\u003e via a simple finger-prick for the thyroid and cardiometabolic panels, following the detailed instructions. Morning saliva should be collected before 9am for optimal cortisol accuracy.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003ePost your samples to the lab\u003c\/strong\u003e using the return instructions in your kit.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eReceive your results within 3–5 working days\u003c\/strong\u003e with specialist commentary and clear next-step guidance.\u003c\/li\u003e\n\u003c\/ol\u003e\n\u003chr\u003e\n\u003ch2\u003eWhat Your Report Includes\u003c\/h2\u003e\n\u003cul\u003e\n\u003cli\u003eAll 22 marker results with male-specific reference ranges\u003c\/li\u003e\n\u003cli\u003eVisual charts for all six sex hormones, your full 4-point cortisol daily curve, thyroid panel, and all six cardiometabolic markers\u003c\/li\u003e\n\u003cli\u003eIntegrated clinical interpretation of how your testosterone, oestradiol, progesterone, DHEAS, cortisol, thyroid markers, and cardiometabolic results interact as a single male hormonal-metabolic system\u003c\/li\u003e\n\u003cli\u003eFree testosterone assessment based on bioavailable LCMS measurement\u003c\/li\u003e\n\u003cli\u003eOestradiol-to-testosterone ratio and total oestrogenic load analysis\u003c\/li\u003e\n\u003cli\u003eCortisol-thyroid T3 conversion and Leydig cell interaction assessment\u003c\/li\u003e\n\u003cli\u003eInsulin resistance pattern assessment (fasting insulin + HbA1c)\u003c\/li\u003e\n\u003cli\u003eCardiovascular risk profile in full androgenic and thyroid hormonal context (HDL, triglycerides, cholesterol, hsCRP)\u003c\/li\u003e\n\u003cli\u003eHashimoto’s autoimmunity assessment (TPO + Tg antibodies)\u003c\/li\u003e\n\u003cli\u003eSpecialist commentary by a Hormone Specialist PhD Doctor\u003c\/li\u003e\n\u003cli\u003ePersonalised next-step recommendations\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003chr\u003e\n\u003ch2\u003eFrequently Asked Questions\u003c\/h2\u003e\n\u003ch3\u003eWhat does this test measure?\u003c\/h3\u003e\n\u003cp\u003eOestradiol (E2), Oestriol (E3), Oestrone (E1), Progesterone, Testosterone, DHEAS (saliva LCMS) + 4-point Cortisol (saliva: morning, noon, evening, night) + TSH, Free T3, Free T4, Total T4, TPO Antibodies, Thyroglobulin Antibodies (blood spot) + Fasting Insulin, HbA1c, HDL Cholesterol, Triglycerides, Total Cholesterol, hsCRP (blood spot) — 22 markers in total.\u003c\/p\u003e\n\u003ch3\u003eWhy is this test better than standard NHS blood tests for men?\u003c\/h3\u003e\n\u003cp\u003eStandard NHS testing typically measures total testosterone by immunoassay, uses TSH as the sole thyroid marker, and does not assess cortisol rhythm, thyroid antibodies, fasting insulin, or hsCRP in combination. This test measures free, bioavailable testosterone by LCMS, provides the full 4-point cortisol stress rhythm, screens for Hashimoto’s autoimmunity, and assesses insulin resistance and cardiovascular inflammation — revealing the hormonal, thyroid, and metabolic root causes of symptoms that standard NHS blood panels systematically miss in men.\u003c\/p\u003e\n\u003ch3\u003eWhy is LCMS used for sex hormones in this test?\u003c\/h3\u003e\n\u003cp\u003eLCMS (Liquid Chromatography–Mass Spectrometry) measures the free, bioavailable fraction of hormones — the portion actually reaching cells and driving androgenic function. In men, this distinction is critical: total testosterone measured by immunoassay includes SHBG-bound hormone that is biologically inactive; a man with high SHBG can have normal total testosterone but critically low free testosterone, producing full symptoms of androgen deficiency. LCMS reveals the true androgenic picture that standard blood tests miss.\u003c\/p\u003e\n\u003ch3\u003eCan I use this test if I am on TRT or levothyroxine?\u003c\/h3\u003e\n\u003cp\u003eYes. This test is particularly valuable for men on TRT or thyroid medication who want a complete picture of how their treatment is affecting their entire androgenic, thyroid, and cardiometabolic system. TRT increases aromatisation, can alter SHBG, thyroid binding, HDL, and triglycerides — all directly assessed here. Follow the instructions provided in your kit.\u003c\/p\u003e\n\u003ch3\u003eIs this test suitable for men with Hashimoto’s or suspected thyroid disease?\u003c\/h3\u003e\n\u003cp\u003eYes — this is the most comprehensive thyroid assessment available in an at-home test for men, including both TPO and Tg antibodies for complete Hashimoto’s screening alongside the full thyroid panel, 4-point cortisol (which drives Hashimoto’s activity), and insulin markers (which worsen thyroid autoimmunity) — providing the full context that standard thyroid blood tests cannot.\u003c\/p\u003e\n\u003ch3\u003eWhen will I get my results?\u003c\/h3\u003e\n\u003cp\u003eWithin \u003cstrong\u003e3–5 working days\u003c\/strong\u003e after your samples reach the laboratory.\u003c\/p\u003e\n\u003ch3\u003eAre there hidden costs?\u003c\/h3\u003e\n\u003cp\u003eNo. Laboratory analysis and specialist review are fully included. You are responsible for postage to the lab.\u003c\/p\u003e\n\u003ch3\u003eHow long is the kit valid?\u003c\/h3\u003e\n\u003cp\u003e12 months from purchase.\u003c\/p\u003e\n\u003chr\u003e\n\u003ch2\u003eWhy We Partner with ZRT Laboratory\u003c\/h2\u003e\n\u003cp\u003eAt Hormone Lab UK, every home test kit is analysed by \u003cstrong\u003eZRT Laboratory\u003c\/strong\u003e, a globally recognised, \u003cstrong\u003eCLIA-certified\u003c\/strong\u003e diagnostic and research laboratory specialising in hormone testing, thyroid testing, fertility testing, adrenal function, neurotransmitter analysis, cardiometabolic health, heavy metals, and wellness testing. Founded in 1998 by renowned biochemist and breast cancer researcher \u003cstrong\u003eDr. David Zava, PhD\u003c\/strong\u003e, ZRT has become one of the world’s leading laboratories for advanced functional health testing.\u003c\/p\u003e\n\u003cp\u003eWith \u003cstrong\u003emore than 11 million laboratory tests\u003c\/strong\u003e performed and healthcare professionals in \u003cstrong\u003eover 96 countries\u003c\/strong\u003e relying on its expertise, ZRT is recognised for scientific excellence, analytical accuracy, and continuous innovation. The laboratory pioneered \u003cstrong\u003eat-home saliva hormone testing\u003c\/strong\u003e, introduced the \u003cstrong\u003efirst commercially available dried blood spot (DBS) hormone tests\u003c\/strong\u003e, and developed \u003cstrong\u003eDried Urine Testing (DUTS)\u003c\/strong\u003e, helping make advanced laboratory diagnostics more accessible and clinically meaningful.\u003c\/p\u003e\n\u003cp\u003eEvery sample is analysed using state-of-the-art \u003cstrong\u003eLC-MS\/MS\u003c\/strong\u003e and \u003cstrong\u003eICP-MS\u003c\/strong\u003e technology under rigorous quality control standards. ZRT collaborates with the \u003cstrong\u003eCDC\u003c\/strong\u003e, \u003cstrong\u003eNIH\u003c\/strong\u003e, and leading universities, supporting ongoing research and advances in laboratory medicine.\u003c\/p\u003e","brand":"ZRT Laboratory","offers":[{"title":"Default Title","offer_id":57960745206141,"sku":null,"price":499.0,"currency_code":"GBP","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/1885\/4859\/files\/SalivaandBloodSpotHormoneTestKit.webp?v=1785073903","url":"https:\/\/hormonelab.co.uk\/es\/products\/male-comprehensive-hormone-thyroid-cardiometabolic-test","provider":"Hormone Lab UK","version":"1.0","type":"link"}